« Previous
Next »
Journal of Vascular Surgery
Volume 46, Issue 3
, Pages 449-454
, September 2007
Fibrinogen and high-sensitive C-reactive protein as serologic predictors for perioperative cerebral microembolic lesions after carotid endarterectomy
References
- Beneficial effect of carotid endarterectomy in symptomatic patients with high-grade carotid stenosis (North American Symptomatic Carotid Endarterectomy Trial Collaborators). N Engl J Med. 1991;325:445–453
- Frequency, clinical significance and course of cerebral ischemic events after carotid endarterectomy evaluated by serial diffusion weighted imaging. Eur J Vasc Endovasc Surg. 2004;27:167–171
- Level of fibrinogen and risk of fatal and non-fatal stroke (EUROSTROKE: a collaborative study among research centres in Europe). J Epidemiol Community Health. 2002;56:i14–i18
- . Fibrinogen predicts ischaemic stroke and advanced atherosclerosis but not echolucent, rupture-prone carotid plaques: the Copenhagen City Heart Study. Eur Heart J. 2003;24:567–576
- Marked decrease in the levels of two inflammatory markers, hs-C-reactive protein and fibrinogen in patients with severe carotid atherosclerosis after eversion carotid endarterectomy. Inflamm Res. 2004;53:631–635
- . Measuring plasma fibrinogen to predict stroke and myocardial infarction: an update. Arterioscler Thromb Vasc Biol. 1999;19:1368–1377
- . Fibrin and its derivatives in the normal and diseased vessel wall. Ann N Y Acad Sci. 1992;667:112–126
- Hyperfibrinogenemia is associated with specific histocytological composition and complications of atherosclerotic carotid plaques in patients affected by transient ischemic attacks. Circulation. 2000;101:744–750
- . Stability of novel plasma markers associated with cardiovascular disease: processing within 36 hours of specimen collection. Clin Chem. 2002;48:1781–1784
- Associations of soluble intercellular adhesion molecule-1 with carotid atherosclerosis progression. Atherosclerosis. 2005;179:155–160
- C-reactive protein is elevated in symptomatic compared with asymptomatic patients with carotid artery disease. Eur J Vasc Endovasc Surg. 2002;23:505–509
- . Association of high sensitivity C-reactive protein with coronary heart disease prediction, but not with carotid atherosclerosis, in patients with hypertension. Circ J. 2004;68:297–303
- . Time-dependent changes of hs-CRP serum concentration in patients with non-ST elevation acute coronary syndrome. Herz. 2004;29:795–801
- . High-sensitivity C-reactive protein in high-grade carotid stenosis: risk marker for unstable carotid plaque. J Vasc Surg. 2003;38:1018–1024
- . C-reactive protein-mediated low density lipoprotein uptake by macrophages: implications for atherosclerosis. Circulation. 2001;103:1194–1197
- . Direct proinflammatory effect of C-reactive protein on human endothelial cells. Circulation. 2000;102:2165–2168
- C-reactive protein is an independent predictor of the rate of increase in early carotid atherosclerosis. Circulation. 2001;104:63–67
- C-reactive protein as a predictor of incident ischemic stroke among patients with preexisting cardiovascular disease. Stroke. 2006;37:1720–1724
- . A prospective study of fibrinogen and risk of myocardial infarction in the Physicians’ Health Study. J Am Coll Cardiol. 1999;33:1347–1352
- Fibrinogen as a predictor of mortality after acute myocardial infarction: a forty-two-month follow-up study. Ital Heart J. 2005;6:315–322
- Role of fibrinogen levels and factor XIII V34L polymorphism in thrombolytic therapy in stroke patients. Stroke. 2006;37:2288–2293
- . Prognostic influence of increased C-reactive protein and fibrinogen levels in ischemic stroke. Stroke. 2001;32:133–138
- . Atherosclerosis–an inflammatory disease. N Engl J Med. 1999;340:115–126
- . Fibrinogen and silent atherosclerosis in subjects with cardiovascular risk factors. Arterioscler Thromb Vasc Biol. 1995;15:1263–1268
- . Angiotensinogen mutations and risk for ischemic heart disease, myocardial infarction, and ischemic cerebrovascular disease (Six case-control studies from the Copenhagen City Heart Study). Ann Intern Med. 2001;134:941–954
Competition of interest: none.
Financial support for this study was provided by the Commission of Clinical Research, Rechts der Isar Medical Center, Technical University of Munich (Kommission für Klinische Forschung, KKF-Nr.: 8744652).
PII: S0741-5214(07)00963-9
doi: 10.1016/j.jvs.2007.05.035
© 2007 The Society for Vascular Surgery. Published by Elsevier Inc. All rights reserved.
« Previous
Next »
Journal of Vascular Surgery
Volume 46, Issue 3
, Pages 449-454
, September 2007
