Journal of Vascular Surgery
Volume 45, Issue 6, Supplement , Pages A64-A73 , June 2007

The role of nitric oxide in the pathophysiology of intimal hyperplasia

Received 18 January 2007 ,Accepted 11 February 2007.

  • Image Result

    Production of nitric oxide (NO) by nitric oxide synthase (NOS). NADP+, nicotinamide adenine dinucleotide phosphate; FAD, flavin adenine dinucleotide; FMN, flavin mononucleotide; HEME, hemoglobin; H4B,

    Production of nitric oxide (NO) by nitric oxide synthase (NOS). NADP+, nicotinamide adenine dinucleotide phosphate; FAD, flavin adenine dinucleotide; FMN, flavin mononucleotide; HEME, hemoglobin; H4B, tetrahydrobiopterin; O2, oxygen; Ca2+, calcium; CaM, calmodulin.

  • Image Result
    Role of nitric oxide (NO) in neointimal hyperplasia. Nitric oxide works to both stimulate (below) and inhibit (above) elements of the vasculature that ultimately results in overall inhibition of neoin

    Role of nitric oxide (NO) in neointimal hyperplasia. Nitric oxide works to both stimulate (below) and inhibit (above) elements of the vasculature that ultimately results in overall inhibition of neointimal hyperplasia. VSMC, vascular smooth muscle cells.

 Competition of interest: Dr Kibbe is a paid carotid stenting proctor for Abbott.

PII: S0741-5214(07)00313-8

doi: 10.1016/j.jvs.2007.02.027

Journal of Vascular Surgery
Volume 45, Issue 6, Supplement , Pages A64-A73 , June 2007

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